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  3. SCSB Colloquium Series with Dr. X. Shawn Liu: Decoding the Epigenome in Brain Function and Disorders
SCSB Colloquium Series with Dr. X. Shawn Liu: Decoding the Epigenome in Brain Function and Disorders
Simons Center for the Social Brain

SCSB Colloquium Series with Dr. X. Shawn Liu: Decoding the Epigenome in Brain Function and Disorders

Add to CalendarAmerica/New_YorkSCSB Colloquium Series with Dr. X. Shawn Liu: Decoding the Epigenome in Brain Function and Disorders05/21/2025 4:00 pm05/21/2025 5:00 pmBuilding 46,46-3002, Singleton Auditorium
May 21, 2025
4:00 pm - 5:00 pm
Location
Building 46,46-3002, Singleton Auditorium
Contact
asokhina@mit.edu
    Description

    Date: Wednesday, May 21, 2025
    Location: 46-3002 (Singleton Auditorium)

    Speaker: X. Shawn Liu, PhD
    Affiliation: Joan and Paul Marks, MD '49 Assistant Professor, Columbia University, Department of Physiology and Cellular Biophysics

    Host: Dr. Rudolf Jaenisch

    Talk title: Decoding the Epigenome in Brain Function and Disorders

    Abstract: Epigenetics is a fundamental mechanism enabling our genome to integrate nature and nurture. It plays a critical role during brain development and perturbations of this mechanism can result in numerous brain disorders. Our laboratory aims to decode the epigenome to understand how genetic information is read in normal physiology and manipulated in diseases. We have developed a series of epigenome editing tools to help dissect the functional significance of epigenetic events. We are combining these molecular tools with genetic and genomic approaches to explore epigenetic mechanisms underlying normal brain functions, and to uncover the epigenetic basis of diseases to accelerate the development of therapeutics. We mainly use human ESC- or iPSC-based system and genetically engineered mouse models to tackle these questions. One of our recent projects revealed the function of DNA methylation in fear memory engram in the basolateral amygdala. Another project demonstrated a methylation editing-based strategy for the treatment of ALS/FTD caused by GGGGCC repeat expansion.

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